erraticphenomenon:

This is the one photo from my Photography/Digital Art class in school that I’ll never forget.It is of my friend’s sister, Paige, who has Williams syndrome. I hope that people can really see the beauty behind this photo. (Click to be redirected to the flickr acct.. this photo is under favorites)

erraticphenomenon:

This is the one photo from my Photography/Digital Art class in school that I’ll never forget.

It is of my friend’s sister, Paige, who has Williams syndrome. I hope that people can really see the beauty behind this photo.

(Click to be redirected to the flickr acct.. this photo is under favorites)


erraticphenomenon:

This is the one photo from my Photography/Digital Art class in school that I’ll never forget.It is of my friend’s sister, Paige, who has Williams syndrome. I hope that people can really see the beauty behind this photo. (Click to be redirected to the flickr acct.. this photo is under favorites)

Reblogging this again because I think more people need to see it.

erraticphenomenon:

This is the one photo from my Photography/Digital Art class in school that I’ll never forget.

It is of my friend’s sister, Paige, who has Williams syndrome. I hope that people can really see the beauty behind this photo.

(Click to be redirected to the flickr acct.. this photo is under favorites)

Reblogging this again because I think more people need to see it.


erraticphenomenon:

This, my friends, is my dog Frankie. In a little over two months he’ll be a year old and by July, I will have owned him a year.

He was abandoned outside my work  on a hot summer day at 12 weeks old in the cage you see in the second picture. I can’t believe he was abandoned, besides my childhood dog, he is the best dog ever. I can’t help but thank the person who left him behind for giving me such a great  companion.

That’s my boy :)


allcreatures:


An angry lioness launches herself at a male - but ends up sitting on his head, looking like a lion hat. She pounced at the male when he tried to discipline her cubs, but misjudged the distance. Park ranger Jacques Matthysen photographed the moment at a game reserve in South Africa.

Picture: JACQUES MATTHYSEN / CATERS NEWS (via Pictures of the day: 27 November 2012 - Telegraph)

allcreatures:

An angry lioness launches herself at a male - but ends up sitting on his head, looking like a lion hat. She pounced at the male when he tried to discipline her cubs, but misjudged the distance. Park ranger Jacques Matthysen photographed the moment at a game reserve in South Africa.

Picture: JACQUES MATTHYSEN / CATERS NEWS (via Pictures of the day: 27 November 2012 - Telegraph)

(via thingswilllookbetterinthemorning)


futureveterinarian:

Slipped Disk Overview
The disks are protective shock-absorbing pads between the bones of the spine (vertebrae). The disks of the spine are also referred to as intervertebral disks. Although they do not actually “slip,” a disk may move, split, or rupture. This can cause the disk cartilage and nearby tissue to fail (herniate), allowing the inner gel portion of the disk to escape into the surrounding tissue. This leaking jelly-like substance can place pressure on the spinal cord or on an adjacent nerve to cause symptoms of pain, numbness, or weakness either around the damaged disk or anywhere along the area supplied by that nerve.
Many people experience no symptoms from a herniated disk, and the majority of people who have herniated disks do not need surgery.
The layman’s term “slipped disk” is, therefore, a misnomer and actually refers to a condition whereby portions of an abnormal, injured, or degenerated disk have protruded against adjacent nerve tissues. This condition is also known as a herniated disk, ruptured disk, or prolapsed disk. The most frequently affected area is in the low back, but any disk can rupture, including those in the neck.
Cross-section (side view picture) of herniated disk between L4 and L5 (the forth and fifth lumbar vertebrae)

futureveterinarian:

Slipped Disk Overview

The disks are protective shock-absorbing pads between the bones of the spine (vertebrae). The disks of the spine are also referred to as intervertebral disks. Although they do not actually “slip,” a disk may move, split, or rupture. This can cause the disk cartilage and nearby tissue to fail (herniate), allowing the inner gel portion of the disk to escape into the surrounding tissue. This leaking jelly-like substance can place pressure on the spinal cord or on an adjacent nerve to cause symptoms of pain, numbness, or weakness either around the damaged disk or anywhere along the area supplied by that nerve.

Many people experience no symptoms from a herniated disk, and the majority of people who have herniated disks do not need surgery.

The layman’s term “slipped disk” is, therefore, a misnomer and actually refers to a condition whereby portions of an abnormal, injured, or degenerated disk have protruded against adjacent nerve tissues. This condition is also known as a herniated disk, ruptured disk, or prolapsed disk. The most frequently affected area is in the low back, but any disk can rupture, including those in the neck.

Cross-section (side view picture) of herniated disk between L4 and L5 (the forth and fifth lumbar vertebrae)


eyes8dow:

kirstenkupcake:

songofthestarwhale:


novagasms:




sometimes the most innocent of children, have answers for the most complicated of questions.

my next thought was the fact that we’re living longer. 

eyes8dow:

kirstenkupcake:

songofthestarwhale:

novagasms:

image


sometimes the most innocent of children, have answers for the most complicated of questions.

my next thought was the fact that we’re living longer. 

(via thepierisfartherthanitseems)


vetstudent-microbiologymaniac:

Equine fetus of approximately 3 months of age. The allantochorion has been incised to expose the fetus enclosed within the almost transparent amnion. Note the umbilical cord at the bottom of the picture and the tortuous blood vessels on the surface of the amnion.
(Arthur’s Veterinary Reproduction and Obstetrics)

vetstudent-microbiologymaniac:

Equine fetus of approximately 3 months of age. The allantochorion has been incised to expose the fetus enclosed within the almost transparent amnion. Note the umbilical cord at the bottom of the picture and the tortuous blood vessels on the surface of the amnion.

(Arthur’s Veterinary Reproduction and Obstetrics)

(via zoodvminthemaking)


erraticphenomenon:

Beautiful. Play on repeat.


Milo Greene - Autumn Tree


futureveterinarian:

The main effect of hyperparathyroidism is hypercalcaemia which causes a range of clinical signs. Polydipsia, polyuria, anorexia, lethargy and depression are the most common signs but animals may also be constipated, weak, stiff-gaited, shivering and vomiting. Mild hypercalcaemia may not generate any overt clinical signs.Sequelae of hyperparathyroidism include fibrous osteodystrophy and organ failure due to metastatic calcification.Osteodystrophy is the osteoclastic resorption of bone and replacement by weaker fibrous tissue. When this occurs in the long bones it causes shifting lameness and weakened bones that are prone to fracture. Compression fractures may also occur spontaneously and if this occurs in the vertebrae, nerve dysfunction results.[2] Weakened bones may also cause tendon and ligament avulsions.Fibrous osteodystrophy in the flat bones of the skull and face causes facial hyperostosis. This is seen in Bran disease or grain overload in horses and also in dogs with primary hyperparathyroidism. The face and head become grossly disfigured by excessive amounts of fibrous tissue laid down in an attempt to consolidate the weakened lamellar bone. In advanced cases, the mandible may become pliant and teeth may loosen, hence the colloquial name, “rubber jaw”. This may interfere with mastication and cause pain, dysphagia and consequent weight loss.Grain overload is also an important cause of severe colic in horses. The sudden increase in fermentation results in enodtoxaemia and acidosis which can be fatal.Animals affected by secondary renal HPT may exhibit classical signs of renal insufficiency such as polydipsia, polyuria, weight loss, vomiting and dehydration.

futureveterinarian:

The main effect of hyperparathyroidism is hypercalcaemia which causes a range of clinical signs. Polydipsia, polyuria, anorexia, lethargy and depression are the most common signs but animals may also be constipated, weak, stiff-gaited, shivering and vomiting. Mild hypercalcaemia may not generate any overt clinical signs.
Sequelae of hyperparathyroidism include fibrous osteodystrophy and organ failure due to metastatic calcification.
Osteodystrophy is the osteoclastic resorption of bone and replacement by weaker fibrous tissue. When this occurs in the long bones it causes shifting lameness and weakened bones that are prone to fracture. Compression fractures may also occur spontaneously and if this occurs in the vertebrae, nerve dysfunction results.[2] Weakened bones may also cause tendon and ligament avulsions.
Fibrous osteodystrophy in the flat bones of the skull and face causes facial hyperostosis. This is seen in Bran disease or grain overload in horses and also in dogs with primary hyperparathyroidism. The face and head become grossly disfigured by excessive amounts of fibrous tissue laid down in an attempt to consolidate the weakened lamellar bone. In advanced cases, the mandible may become pliant and teeth may loosen, hence the colloquial name, “rubber jaw”. This may interfere with mastication and cause pain, dysphagia and consequent weight loss.
Grain overload is also an important cause of severe colic in horses. The sudden increase in fermentation results in enodtoxaemia and acidosis which can be fatal.
Animals affected by secondary renal HPT may exhibit classical signs of renal insufficiency such as polydipsia, polyuria, weight loss, vomiting and dehydration.